Human mast cells capture, store, and release bioactive, exogenous IL-17A.

نویسندگان

  • Troy Noordenbos
  • Iris Blijdorp
  • Sijia Chen
  • Jan Stap
  • Erik Mul
  • Juan D Cañete
  • Erik Lubberts
  • Nataliya Yeremenko
  • Dominique Baeten
چکیده

IL-17A, a major proinflammatory cytokine, can be produced by a variety of leukocytes, but its exact cellular source in human inflammatory diseases remains incompletely understood. IL-17A protein is abundantly found in mast cells in human tissues, such as inflamed synovium, but surprisingly, mechanistic murine studies failed to demonstrate IL-17A production by mast cells. Here, we demonstrate that primary human tissue mast cells do not produce IL-17A themselves but actively capture exogenous IL-17A through receptor-mediated endocytosis. The exogenous IL-17A is stored in intracellular granules and can subsequently be released in a bioactive form. This novel mechanism confers to mast cells the capacity to steer IL-17A-mediated tissue inflammation by the rapid release of preformed cytokine.

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عنوان ژورنال:
  • Journal of leukocyte biology

دوره 100 3  شماره 

صفحات  -

تاریخ انتشار 2016